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Exam Number : C2150-630
Exam Name : IBM BigFix Inventory V9.5 and Licence Metric Tool V9.2 Administration
Vendor Name : IBM
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This intermediate level certification is for administrators who are familiar with product functionality and have extensive hands-on experience with IBM BigFix Inventory V9.5 and License Metric Tool V9.2. They plan, install, configure, deploy, maintain, troubleshoot and support it.
The key areas of competency are:
Describe BigFix architecture and Console.
Design IBM BigFix Inventory V9.5 and License Metric Tool V9.2 solutions based on customer environment and requirements.
Install and configure IBM BigFix Inventory V9.5 and License Metric Tool V9.2 software and components.
Perform problem determination and performance tuning for IBM BigFix Inventory V9.5 and License Metric Tool V9.2
Customize various components of IBM BigFix Inventory V9.5 and License Metric Tool V9.2 to support customer requirements.
Troubleshoot, maintain and optimize the IBM BigFix Inventory V9.5 and License Metric Tool V9.2 environment
It is expected that these administrators are generally self-sufficient and are able to perform the tasks involved in the job role with limited assistance from peers, product documentation, and vendor support services.
Basic knowledge of LDAP including Windows Active Directory.
Working knowledge of:
TCP/IP, network topology and troubleshooting.
current Microsoft SQL and IBM DB2 Server.
unattended installation methods for software products.
Linux and Windows operating systems and software installation.
virtualization technologies.



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alphaB-crystallin Is a Marker of Aggressive Breast melanoma behavior however doesn't Independently Predict for patient effect | C2150-630 study guide and PDF Dumps

strategies sufferers and Tissues

The HE10/97 trial[30] changed into a randomized section III trial (ACTRN12611000506998) in patients with excessive-chance node-bad or intermediate/excessive-risk node-effective operable breast cancer, evaluating four cycles of epirubicin (E) adopted via four cycles of intensified CMF (E-CMF) with three cycles of E, adopted by way of three cycles of paclitaxel (T, Taxol®, Bristol Myers-Squibb, Princeton, NJ) adopted by means of three cycles of intensified CMF (E-T-CMF). All cycles got every two weeks with G-CSF support. Dose intensity of all medication in each medication fingers became identical, however cumulative doses and length of chemotherapy duration differed. absolutely, 595 eligible sufferers entered the study in a length of 3.5 years (1997–2000).

The HE10/00 trial[31,32] changed into a randomized part III trial (ACTRN12609001036202) during which sufferers were handled with E-T-CMF (precisely as within the HE10/97 trial) or with 4 cycles of epirubicin/paclitaxel (ET) aggregate (given on the identical day) each three weeks adopted by way of three cycles of intensified CMF every two weeks (ET-CMF). by using examine design, the cumulative doses and the chemotherapy length have been identical in the two fingers however dose intensity of epirubicin and paclitaxel was double within the E-T-CMF arm. a complete of 1086 eligible patients with node-high-quality operable breast melanoma had been accrued in a length of 5 years (2000–2005).

medication schedules for the two experiences, baseline characteristics and clinical consequences of both trials have already been described in element.[30–33] scientific protocols were permitted via local regulatory authorities, while the current translational analysis protocol turned into authorized through the Bioethics Committee of the Aristotle university of Thessaloniki, school of medicine, beneath the typical title "Molecular investigation of the predictive and/or prognostic role of crucial sign transduction pathways in breast melanoma" (A7150/18-three-2008). All sufferers signed a examine-certain written counseled consent earlier than randomization, which apart from presenting consent for the trial allowed using their biological cloth for future analysis applications.

In complete, 271 sufferers from the HE10/ninety seven trial and 669 from the HE10/00 trial were covered in this analyze, in accordance with tissue availability. fundamental tumor diameter, axillary nodal repute and tumor grade were got from the pathology report in each case.

TMA building

Formalin-fixed paraffin-embedded (FFPE) tissue samples from patient tumors (paraffin blocks) were collected retrospectively within the first trial (HE10/97) and prospectively in the 2nd (HE10/00). The present study became carried out on tissue microarrays (TMAs). representative hematoxylin-eosin stained sections from the tissue blocks had been reviewed with the aid of two experienced in breast melanoma pathologists and essentially the most consultant tumor areas have been marked for the development of ΤΜΑ blocks with a guide arrayer (mannequin I, Beecher contraptions, San Prairie, WI). each and every tumor turned into represented by means of 2 tissue cores, 1.5 mm in diameter, that have been got from distinct and most representative areas of simple invasive or in some circumstances from synchronous axillary lymph node metastases and re-embedded in recipient paraffin blocks. TMAs additionally contained control cores from different tumors and non-neoplastic tissues.[32] In total, 51 TMAs had been created. situations not represented, broken or inadequate on the TMA sections had been re-cut from the normal blocks, the place tissue material was nevertheless obtainable.

Immunohistochemistry (IHC) for Breast cancer Subtyping

IHC for ER (clone 6 F11, Leica Biosystems, Newcastle Upon Tyne, UK), PgR (clone 1A6, Leica Biosystems), HER2 (pl, code A0485, Dako, Glostrup, DK), Ki67 (clone MIB1, Dako), CK5 (clone XM26, Leica Biosystems), CK14 (clone LL002, Leica Biosystems), CK17 (E31, Leica Biosystems) and EGFR (clone 31G7, Invitrogen, Carlsbad, CA) turned into carried out on serial 2 micron thick sections, the usage of the Bond Max and Bond III autostainers (Leica Microsystems, Wetzlar, Germany), as previously described.[34] IHC became carried out centrally at the Laboratory of Molecular Oncology of the Hellenic basis for cancer analysis, Aristotle university of Thessaloniki school of drugs. Vimentin (clone V9, Dako) and cytokeratin 8/18 (clone 5D3, Leica Biosystems) have been used as immunoreactivity controls and for the identification of tumor cells. Tissue samples poor with the above two antibodies were excluded from the look at. The contrast of all IHC sections turned into completed by means of experienced breast cancer pathologists, blinded to the affected person clinical qualities and survival information.

All tumors protected during this look at had been categorized in accordance with ER, PgR, HER2, Ki67, CK5 and EGFR, as Luminal A (ER-fantastic and/or PgR-high-quality, HER2-negative and Ki67low), Luminal B (ER-positive and/or PgR-tremendous, HER2-terrible and Ki67high), luminal-HER2 (ER-tremendous and/or PgR-fine and HER2-positive), HER2-enriched (ER-bad, PgR-terrible, HER2-effective) and TNBC (ER-negative, PgR-poor, HER2-negative). Tumors had been categorised as Ki67high when ≥14% of neoplastic cells had been tremendous.[35] TNBC of the basal core phenotype (BCP) have been additionally unique as CK5-advantageous and/or EGFR-tremendous.

IHC for alphaB-crystallin, BRCA1 and p53 Markers

The IHC formula become performed the usage of the Bond Max and Bond III autostainers (Leica Microsystems). The Mouse IgG1 monoclonal antibody, clone 1B6.1-3G4 (Stressgen Biotechnologies, San Diego, CA) was used for the detection of full-size alphaB-crystallin (1:200 dilution, 1 h incubation at room temperature). The MS110 antibody from Merck KGaA (Darmstadt, Germany) was used for BRCA1 detection (1:one hundred fifty dilution, for 20 min), while p53 protein was detected with the DO-7 clone (Dako) at dilution 1:one hundred, for 20 min. The antigen–antibody complex changed into visualized the use of DAB as a chromogen. Slides have been counterstained with Mayer's hematoxylin for 10 min (Leica), washed in water, dehydrated and mounted.

IHC comparison

ER, PgR, HER2, Ki67 and EGFR protein expression become evaluated in line with the dependent or proposed criteria.[35–38] CK5, CK14 and CK17 expression changed into regarded as bad (absence of staining) or effective (any cytoplasmic staining of tumor cells).[34] For alphaB-crystallin the percent of advantageous tumor cells and the depth (light, moderate, effective) had been recorded in each case. The distribution of continuous positivity values revealed a natural cut-off at 30%. Tumors had been considered poor, when no certain cytoplasmic staining was observed, weakly fantastic (<30% wonderful neoplastic cells) and strongly high-quality (≥30% nice neoplastic cells). within the latter category staining depth was predominantly strong; for this reason, depth become not included within the statistical analysis. The above staining demo changed into in accordance with Moyano's outdated file,[7] who used a cutoff of ≥30% to consider low and excessive alphaB-crystallin expressing tumors. BRCA1 staining turned into evaluated by using the histological ranking (H-ranking) at a positivity reduce-off of > a hundred.[39] For p53, ≥10% nuclear staining of invasive cancer cells changed into regarded positive.[40] IHC positivity standards for all antibodies are proven in desk 1 .

HER2 popularity became also investigated in all instances with FISH the use of the ZytoLightH SPEC HER2/TOP2A/CEN17 triple color probe (ZytoVision, Bremerhaven, Germany), as up to now described.[41]

BRCA Mutations

DNA for BRCA screening was purchasable for 127 of the 940 patients protected within the present analysis. Seventy-nine patients have been screened for BRCA1 simplest, three patients were screened for BRCA2 only and seven patients have been screened in each genes. Thirty-eight patients had been now not screened due to low quality of the DNA pattern. Genomic DNA was remoted from peripheral blood lymphocytes following the salt extraction manner.[42] The amount and pleasant of the DNA samples were determined by UV absorbance the use of a Nanodrop™ 1000 (Thermo Fisher Scientific, MA) and agarose gel electrophoresis. BRCA1 and BRCA2 had been amplified the usage of intronic primer pairs flanking each exon and three diagnostic PCR reactions were additionally carried out to be able to realize the Greek founder genomic rearrangements involving exons 20, 23 and 24.[43] PCR amplifications were performed in a Veriti ninety six-neatly Thermal Cycler and the PCR items have been directly sequenced the use of the v.three.1 BigDye Terminator Cycle Sequencing kit on an 3130XL Genetic Analyzer (all three from utilized Biosystems, Foster metropolis, CA), in response to the brand's guidelines. In some circumstances of excessive-possibility households, genomic DNA become also examined through MLPA evaluation (MRC-Holland). Sequence variations, apart from widely wide-spread polymorphisms, were established in an impartial blood demo by way of sequencing each ahead and reverse instructions. All nucleotide numbers check with the wild-type genomic DNA sequence of BRCA1 NG_005905.2 and BRCA2 NG_012772.1, as said in RefSeqGene data. Primer sequences and protocols can be found upon request.

Statistical evaluation

specific facts are displayed as frequencies and corresponding percentages, whereas continuous statistics by way of median and latitude. Comparisons of express facts between corporations have been carried out with the aid of Fisher's accurate or Pearson chi-rectangular assessments. For continual records, the assumptions for performing parametric assessments had been not fulfilled (the statistics have been not perpetually dispensed); hence the non-parametric Kruskal-Wallis check became used. For numerical ordinal information, the Jonckheere-Terpstra fashion examine (JT examine) changed into carried out. sickness-free survival (DFS) become measured from the date of randomization except tumor recurrence, secondary neoplasm or dying from any cause. universal survival (OS) became measured from the date of randomization until loss of life from any trigger. Surviving sufferers have been censored on the date of ultimate contact. Time-to-event distributions have been offered the usage of Kaplan-Meier curves and in comparison the use of the log-rank check.

Univariate Cox regression analyses have been carried out for OS and DFS, to determine the prognostic or predictive magnitude in paclitaxel medication of the examined biomarkers. A backward selection manner with a removal criterion of p > 0.10 become performed in the multivariate Cox regression analysis with a view to determine enormous components among the following: randomization group (ET-CMF, E-CMF, vs. E-C-MF), involved axillary lymph nodes (≥4 vs. 0–three), tumor grade (III-Undifferentiated vs. I-II), tumor size (>5 cm, 2–5 cm vs. ≤2 cm), category of surgical procedure (breast conserving surgery vs. modified radical mastectomy, ΜRΜ), histological classification (ductal vs. different) and adjuvant hormonal remedy (yes, lacking vs. no). The examined markers had been included in the closing mannequin the use of the categorization: alphaB-crystallin (powerful high-quality, susceptible advantageous vs. poor), p53 (nice vs. terrible) and BRCA1 (effective vs. poor).

outcomes of this study have been offered based on stated innovations for tumor marker prognostic stories.[44] The design of the analyze is prospective-retrospective, as up to now described by Simon et al..[45] All statistical tests have been two sided and p < 0.05 become regarded statistically gigantic. No alterations for varied exams are mentioned. The statistical evaluation became performed using right here statistical software: SPSS for windows (version 15.0, IBM company, ny) and SAS (edition 9.three, SAS Institute Inc., Cary, NC).




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